Articles

Articles 28 registres trobats  inicianterior12 - 21següent  anar al registre: La cerca s'ha fet en 0.02 segons. 
12.
14 p, 2.6 MB Repurposed analog of GLP-1 ameliorates hyperglycemia in type 1 diabetic mice through pancreatic cell reprogramming / Villalba Felipe, Adrián (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez Fernández, Silvia (Institut Germans Trias i Pujol) ; Perna-Barrull, David (Institut Germans Trias i Pujol) ; Ampudia Carrasco, Rosa María (Institut Germans Trias i Pujol) ; Gómez Muñoz, Laia (Institut Germans Trias i Pujol) ; Pujol-Autonell, Irma (Institut Germans Trias i Pujol) ; Aguilera, Eva (Institut Germans Trias i Pujol) ; Coma, Mireia (Anaxomics Biotech SL) ; Cano-Sarabia, Mary (Institut Català de Nanociència i Nanotecnologia) ; Vázquez, Federico (Institut Germans Trias i Pujol) ; Verdaguer, Joan (Universitat de Lleida. Departament de Medicina Experimental) ; Vives Pi, Marta (Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas)
Type 1 diabetes is an autoimmune disease caused by the destruction of the insulin-producing β-cells. An ideal immunotherapy should combine the blockade of the autoimmune response with the recovery of functional target cell mass. [...]
2020 - 10.3389/fendo.2020.00258
Frontiers in Endocrinology, Vol. 11 (May 2020) , art. 258  
13.
15 p, 4.1 MB Prenatal Betamethasone interferes with immune system development and alters target cells in autoimmune diabetes / Perna-Barrull, David (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Fernández, Silvia (Institut Germans Trias i Pujol) ; Pujol-Autonell, Irma (Institut Germans Trias i Pujol) ; Gieras, A. (Department of Immunology. University Medical Center Hamburg-Eppendorf) ; Ampudia Carrasco, Rosa María (Institut Germans Trias i Pujol) ; Villalba Felipe, Adrián (Institut Germans Trias i Pujol) ; Glau, L. (Department of Immunology. University Medical Center Hamburg-Eppendorf) ; Tolosa, E. (Department of Immunology. University Medical Center Hamburg-Eppendorf) ; Vives Pi, Marta (Institut Germans Trias i Pujol) ; Universitat Autònoma de Barcelona
Non-genetic factors are crucial in the pathogenesis of type 1 diabetes (T1D), a disease caused by autoimmunity against insulin-producing β-cells. Exposure to medications in the prenatal period may influence the immune system maturation, thus altering self-tolerance. [...]
2019 - 10.1038/s41598-018-37878-9
Scientific reports, Vol. 9 Núm. 1 (january 2019) , p. 1235  
14.
18 p, 2.6 MB Partial remission and early stages of pediatric type 1 diabetes display immunoregulatory changes. A pilot study / Villalba Felipe, Adrián (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Fonolleda, Mireia (Institut Germans Trias i Pujol) ; Murillo-Vallés, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Fernández, Silvia (Institut Germans Trias i Pujol) ; Ampudia Carrasco, Rosa María (Institut Germans Trias i Pujol) ; Perna-Barrull, David (Institut Germans Trias i Pujol) ; Raina, María Belén (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Quirant, Bibiana (Institut Germans Trias i Pujol) ; Planas, Raquel (Institut Germans Trias i Pujol) ; Teniente Serra, Aina (Institut Germans Trias i Pujol) ; Bel, Joan (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vives Pi, Marta (Institut Germans Trias i Pujol) ; Universitat Autònoma de Barcelona
Type 1 diabetes (T1D) is a chronic metabolic disease of unknown etiology that results from β-cell destruction. The onset of the disease, which arises after a long asymptomatic period of autoimmune attack, may be followed by a relapsing and remitting progression, a phenomenon that is most evident during the partial remission phase (PR). [...]
2019 - 10.1016/j.trsl.2019.03.002
Translational Research, Vol. 210 (august 2019) , p. 8-25  
15.
11 p, 5.0 MB B-Lymphocyte Phenotype Determines T-Lymphocyte Subset Differentiation in Autoimmune Diabetes / Egia-Mendikute, Leire (Institut de Recerca Biomèdica de Lleida) ; Arpa, Berta (Institut de Recerca Biomèdica de Lleida) ; Rosell-Mases, Estela (Institut de Recerca Biomèdica de Lleida) ; Corral-Pujol, Marta (Institut de Recerca Biomèdica de Lleida) ; Carrascal, Jorge (Institut de Recerca Biomèdica de Lleida) ; Carrillo, Jorge (Institut de Recerca Biomèdica de Lleida) ; Mora, Conchi (Institut de Recerca Biomèdica de Lleida) ; Chapman, Harold (Jackson Laboratory) ; Panosa, Anaïs (Institut de Recerca Biomèdica de Lleida) ; Vives Pi, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Stratmann, Thomas (Universitat de Barcelona. Departament de Biologia Cel·lular, Fisiologia i Immunologia) ; Serreze, David (Jackson Laboratory) ; Verdaguer, Joan (Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas)
Previous studies indicate that B-lymphocytes play a key role activating diabetogenic T-lymphocytes during the development of autoimmune diabetes. Recently, two transgenic NOD mouse models were generated: the NOD-PerIg and the 116C-NOD mice. [...]
2019 - 10.3389/fimmu.2019.01732
Frontiers in immunology, Vol. 10 (2019) , p. 1732  
16.
13 p, 1.5 MB Impaired Phagocytosis in Dendritic Cells From Pediatric Patients With Type 1 Diabetes Does Not Hamper Their Tolerogenic Potential / Rodríguez Fernández, Silvia (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Murillo-Vallés, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Villalba Felipe, Adrián (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Perna-Barrull, David (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Cano-Sarabia, Mary (Institut Català de Nanociència i Nanotecnologia) ; Gómez Muñoz, Laia (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Aguilera, Eva (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Maspoch Comamala, Daniel (Institut Català de Nanociència i Nanotecnologia) ; Vázquez, Federico (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Bel, Joan (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vives Pi, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol)
Type 1 diabetes (T1D) is prompted by defective immunological tolerance, an event in which dendritic cells (DCs) are crucial as immune response orchestrators. In fact, they contribute to maintaining tolerance to self-antigens, but they can also prompt an immunogenic response against them, leading to autoimmunity. [...]
2019 - 10.3389/fimmu.2019.02811
Frontiers in immunology, Vol. 10 (November 2019) , art. 2811  
17.
12 p, 2.5 MB Liposome-based immunotherapy against autoimmune diseases : Therapeutic effect on multiple sclerosis / Pujol-Autonell, Irma (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Mansilla Lopez, Maria Jose (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez Fernández, Silvia (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Cano-Sarabia, Mary (Institut Català de Nanociència i Nanotecnologia) ; Navarro-Barriuso, Juan (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Ampudia Carrasco, Rosa María (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rius, Aleix (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; García Jimeno, Sonia (Institut Català de Nanociència i Nanotecnologia) ; Perna-Barrull, David (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Martínez Cáceres, Eva María (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Maspoch Comamala, Daniel (Institut Català de Nanociència i Nanotecnologia) ; Vives Pi, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol)
Based on the ability of apoptosis to induce immunological tolerance, liposomes were generated mimicking apoptotic cells, and they arrest autoimmunity in Type 1 diabetes. Our aim was to validate the immunotherapy in other autoimmune disease: multiple sclerosis. [...]
2017 - 10.2217/nnm-2016-0410
Nanomedicine, Vol. 12, Issue 11 (June 2017) , p. 1231-1242  
18.
3 p, 591.8 KB L'estimulació sensorial millora el dany cerebral en ratolins models de prematurs / Vives Pi, Marta (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia)
Investigadores de l'INc­UAB han demostrat per primera vegada en un model murí de nadons prematurs que una lesió cerebral per hipòxia i isquèmia condiciona de forma diferent les capacitats funcionals neurològiques, cognitives i emocionals depenent del sexe, l'edat i la tasca a realitzar, tot i tenir el mateix grau de severitat neuropatològica en mascles i femelles. [...]
Investigadores del INc-UAB han demostrado por primera vez en un modelo murino de bebés prematuros que una lesión cerebral por hipoxia e isquemia condiciona de forma diferente las capacidades funcionales neurológicas, cognitivas y emocionales dependiendo del sexo, la edad y la tarea a realizar, a pesar de tener el mismo grado de severidad neuropatológica en machos y hembras. [...]
A research conducted by the INc-UAB shows that the same perinatal brain injury caused by hypoxia and ischemia have differentiated effects on each gender, but can be improved through tactile and proprioceptive stimuli. [...]

2019
UAB divulga, Març 2019, p. 1-2
3 documents
19.
15 p, 5.9 MB Prenatal administration of Betamethasone causes changes in the T cell receptor repertoire influencing Development of autoimmunity / Gieras, Anna (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Gehbauer, Christina (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Perna-Barrull, David (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Engler, Jan Broder (Institute of Neuroimmunology and Multiple Sclerosis. Center for Molecular Neurobiology Hamburg) ; Diepenbruck, Ines (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Glau, Laura (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Joosse, Simon A. (University Medical Center Hamburg-Eppendorf. Department of Tumor Biology) ; Kersten, Nora (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Klinge, Stefanie (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Mittrücker, Hans-Willi (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Friese, Manuel A. (Institute of Neuroimmunology and Multiple Sclerosis, Center for Molecular Neurobiology Hamburg) ; Vives Pi, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Tolosa, Eva (University Medical Center Hamburg-Eppendorf. Department of Immunology) ; Universitat Autònoma de Barcelona
Prenatal glucocorticoids are routinely administered to pregnant women at risk of preterm delivery in order to improve survival of the newborn. However, in half of the cases, birth occurs outside the beneficial period for lung development. [...]
2017 - 10.3389/fimmu.2017.01505
Frontiers in immunology, Vol. 8 (november 2017) , p. 1-15  
20.
17 p, 2.4 MB Phosphatidylserine-liposomes promote tolerogenic features on dendritic cells in human type 1 diabetes by apoptotic mimicry / Rodriguez Fernandez, Silvia (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Pujol-Autonell, Irma (Institut Germans Trias i Pujol) ; Brianso, Ferran (Universitat de Barcelona. Departament de Genètica, Microbiologia i Estadística) ; Perna-Barrull, David (Institut Germans Trias i Pujol) ; Cano-Sarabia, Mary (Institut Català de Nanociència i Nanotecnologia) ; García Jimeno, Sonia (Institut Català de Nanociència i Nanotecnologia) ; Villalba Felipe, Adrián (Institut Germans Trias i Pujol) ; Sánchez, Alex (Universitat de Barcelona. Departament de Genètica, Microbiologia i Estadística) ; Aguilera, Eva (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vázquez, Federico (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Verdaguer, Joan (Universitat de Lleida. Departament de Medicina Experimental) ; Maspoch Comamala, Daniel (Institució Catalana de Recerca i Estudis Avançats) ; Vives Pi, Marta (Institut Germans Trias i Pujol)
Type 1 diabetes (T1D) is a metabolic disease caused by the autoimmune destruction of insulin-producing β-cells. With its incidence increasing worldwide, to find a safe approach to permanently cease autoimmunity and allow β-cell recovery has become vital. [...]
2018 - 10.3389/fimmu.2018.00253
Frontiers in immunology, Vol. 9 (Feb. 2018) , art. 253  
21.
8 p, 1.1 MB Expression of REG family genes in human inflammatory bowel diseases and its regulation / Tsuchida, Chikatsugu (Saiseikai Nara Hospital, Japan) ; Sakuramoto-Tsuchida, Sumiyo (Department of Biochemistry, Nara Medical University) ; Taked, Maiko (Department of Laboratory Medicine and Pathology, National Hospital Organization Kinki-chuo Chest Medical Center) ; Itaya-Hironaka, Asako (Department of Biochemistry, Nara Medical University) ; Yamauchi, Akiyo (Department of Biochemistry, Nara Medical University) ; Misu, Masayasu (Department of Biochemistry, Nara Medical University) ; Shobatake, Ryogo (Department of Biochemistry, Nara Medical University) ; Uchiyama, Tomoko (Department of Diagnostic Pathology, Nara Medical University) ; Makino, Mai (Department of Biochemistry, Nara Medical University) ; Pujol-Autonell, Irma (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vives Pi, Marta (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Ohbayashi, Chiho (Department of Diagnostic Pathology, Nara Medical University) ; Takasawa, Shin (Department of Biochemistry, Nara Medical University)
The pathophysiology of inflammatory bowel disease (IBD) reflects a balance between mucosal injury and reparative mechanisms. Some regenerating gene (Reg) family members have been reported to be expressed in Crohn's disease (CD) and ulcerative colitis (UC) and to be involved as proliferative mucosal factors in IBD. [...]
2017 - 10.1016/j.bbrep.2017.10.003
Biochemistry and Biophysics Reports, Vol. 12 (october 2017) , p. 198-205  

Articles : 28 registres trobats   inicianterior12 - 21següent  anar al registre:
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