Articles publicats

Articles publicats 2 registres trobats  La cerca s'ha fet en 0.03 segons. 
1.
14 p, 2.3 MB Degradation of GRK2 and AKT is an early and detrimental event in myocardial ischemia/reperfusion / Penela, Petronila (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares) ; Inserte, Javier (Universitat Autònoma de Barcelona) ; Ramos, Paula (Centro de Biología Molecular Severo Ochoa) ; Rodriguez-Sinovas, Antonio (Universitat Autònoma de Barcelona) ; García-Dorado, David (Universitat Autònoma de Barcelona) ; Mayor, F. (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares)
Background: Identification of signaling pathways altered at early stages after cardiac ischemia/reperfusion (I/R) is crucial to develop timely therapies aimed at reducing I/R injury. The expression of G protein-coupled receptor kinase 2 (GRK2), a key signaling hub, is up-regulated in the long-term in patients and in experimental models of heart failure. [...]
2019 - 10.1016/j.ebiom.2019.09.019
EBioMedicine, Vol. 48 (october 2019) , p. 605-618  
2.
12 p, 3.5 MB A single faecal microbiota transplantation modulates the microbiome and improves clinical manifestations in a rat model of colitis / Lleal, Marina (Hospital Universitari Vall d'Hebron) ; Sarrabayrouse, Guillaume (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Willamil, Joseane (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Santiago, Alba (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Pozuelo, Marta (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Manichanh, Chaysavanh (Centro de Investigación Biomédica en Red de Enfermedades Hepáticas y Digestivas)
Background: Faecal microbiota transplantation (FMT) is a novel potential therapy for inflammatory bowel diseases, but it is poorly characterised. Methods: We evaluated the performance of the mouse and rat as a pre-clinical model for human microbiota engraftment. [...]
2019 - 10.1016/j.ebiom.2019.10.002
EBioMedicine, Vol. 48 (october 2019) , p. 630-641  

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