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Articles, 3 records found
Articles 3 records found  
1.
21 p, 1.0 MB Inhibition of the neuronal NFκB pathway attenuates bortezomib-induced neuropathy in a mouse model / Alé, Albert (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Bruna, Jordi (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Calls, Aina (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Karamita, Maria (Hellenic Pasteur Institute) ; Haralambous, Sylva (Hellenic Pasteur Institute) ; Probert, Lesley (Hellenic Pasteur Institute) ; Navarro, X. (Xavier) (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Udina i Bonet, Esther (Universitat Autònoma de Barcelona. Institut de Neurociències)
© 2016 Elsevier B. V. Bortezomib is a proteasome inhibitor with a remarkable antitumor activity, used in the clinic as first line treatment for multiple myeloma. One hallmark of bortezomib mechanism of action in neoplastic cells is the inhibition of nuclear factor kappa B (NFκB), a transcription factor involved in cell survival and proliferation. [...]
2016 - 10.1016/j.neuro.2016.05.004
NeuroToxicology, Vol. 55 (2016) , p. 58-64  
Detailed record -
2.
28 p, 10.5 MB Treatment with anti-TNF alpha protects against the neuropathy induced by the proteasome inhibitor bortezomib in a mouse model / Alé, Albert (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Bruna, Jordi (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Morell, Marta (Universitat Autònoma de Barcelona. Institut de Neurociències) ; van de Velde, Helgi (Janssen Research and Development) ; Monbaliu, Johan (Janssen Research and Development) ; Navarro, X. (Xavier) (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Udina i Bonet, Esther (Universitat Autònoma de Barcelona. Institut de Neurociències)
Bortezomib (BTZ), a proteasome inhibitor, is an effective anti-neoplastic drug used in the treatment of multiple myeloma and mantle cell lymphoma. However, it can induce a reversible peripheral neuropathy that may lead to treatment discontinuation. [...]
2014 - 10.1016/j.expneurol.2013.12.020
Experimental Neurology, Vol. 253 (march 2014) , p. 165-173  
Detailed record -
3.
11 p, 3.3 MB Differential effects on KCC2 expression and spasticity of ALS and traumatic injuries to motoneurons / Mòdol, Laura (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Mancuso, Renzo (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Alé, Albert (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Francos Quijorna, Isaac (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Navarro, X. (Xavier) (Xavier) (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia)
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease manifested by progressive muscle atrophy and paralysis due to the loss of upper and lower motoneurons (MN). Spasticity appears in ALS patients leading to further disabling consequences. [...]
2014 - 10.3389/fncel.2014.00007
Frontiers in cellular neuroscience, Vol. 8 Núm. 7 (Jan. 2014) , p. 1-11  
Detailed record - Creative Commons

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