Resultados globales: 4 registros encontrados en 0.02 segundos.
Artículos, Encontrados 4 registros
Artículos Encontrados 4 registros  
1.
11 p, 1.0 MB Cardiomyocyte hypertrophy induced by Endonuclease G deficiency requires reactive oxygen radicals accumulation and is inhibitable by the micropeptide humanin / Blasco, Natividad (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain) ; Cámara, Yolanda (Research Group on Neuromuscular and Mitochondrial Diseases, Vall d'Hebron Research Institute, Universitat Autònoma de Barcelona and CIBERER, Barcelona, Spain) ; Núñez, Estefanía (Cardiovascular Proteomics group, Spanish National Center for Cardiovascular Research (CNIC) and CIBERCV Madrid, Spain) ; Beà, Aida (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain) ; Barés, Gisel (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain) ; Forné, Carles (Biostatistics Unit, IRBLleida, Universitat de Lleida, Lleida, Spain) ; Ruíz-Meana, Marisol (Cardiovascular Diseases Research Group, VHIR and CIBERCV, Barcelona, Spain) ; Girón, Cristina (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain) ; Barba, Ignasi (Cardiovascular Diseases Research Group, VHIR and CIBERCV, Barcelona, Spain) ; García-Arumí, Elena (Research Group on Neuromuscular and Mitochondrial Diseases, Vall d'Hebron Research Institute, Universitat Autònoma de Barcelona and CIBERER, Barcelona, Spain) ; García Dorado, David (Cardiovascular Diseases Research Group, VHIR and CIBERCV, Barcelona, Spain) ; Vázquez, Jesús (Cardiovascular Proteomics group, Spanish National Center for Cardiovascular Research (CNIC) and CIBERCV Madrid, Spain) ; Martí Seves, Ramon (Research Group on Neuromuscular and Mitochondrial Diseases, Vall d'Hebron Research Institute, Universitat Autònoma de Barcelona and CIBERER, Barcelona, Spain) ; Llovera, Marta (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain) ; Sanchis, Daniel (Cell Signaling and Apoptosis group, Universitat de Lleida - IRBLleida, Lleida, Spain)
The endonuclease G gene (Endog), which codes for a mitochondrial nuclease, was identified as a determinant of cardiac hypertrophy. How ENDOG controls cardiomyocyte growth is still unknown. Thus, we aimed at finding the link between ENDOG activity and cardiomyocyte growth. [...]
2018 - 10.1016/j.redox.2018.02.021
Redox Biology, Vol. 16 (march 2018) , p. 146-156  
2.
12 p, 2.0 MB The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins / Moubarak, Rana S. (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Solé, Carme (Institut de Recerca Biomèdica de Lleida) ; Pascual, Marta (Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas) ; Gutiérrez, Humberto (Cardiff University. School of Biosciences) ; Llovera, Marta (Institut de Recerca Biomèdica de Lleida) ; Pérez García, M. José (Institut de Recerca Biomèdica de Lleida) ; Gozzelino, Raffaella (Institut de Recerca Biomèdica de Lleida) ; Segura, Miguel F. (Institut de Recerca Biomèdica de Lleida) ; Iglesias Guimarais, Victoria (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Reix, Stéphanie (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Soler, Rosa M. (Institut de Recerca Biomèdica de Lleida) ; Davies, Alun M. (Cardiff University. School of Biosciences) ; Soriano García, Eduardo (Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas) ; Yuste Mateos, Víctor J. (Víctor José) (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Comella i Carnicé, Joan Xavier, 1963- (Universitat Autònoma de Barcelona. Institut de Neurociències)
FLICE-inhibitory protein (FLIP) is an endogenous inhibitor of the signaling pathway triggered by the activation of death receptors. Here, we reveal a novel biological function for the long form of FLIP (FLIP-L) in neuronal differentiation, which can be dissociated from its antiapoptotic role. [...]
2010 - 10.1523/JNEUROSCI.0537-10.2010
The Journal of neuroscience, Vol. 30 Núm. 17 (April 2010) , p. 6094-6105  
3.
13 p, 844.6 KB A TrkB/EphrinA interaction controls retinal axon branching and synaptogenesis / Marler, Katharine J. M. (MRC Centre for Developmental Neurobiology) ; Becker-Barroso, Elena (MRC Centre for Developmental Neurobiology) ; Martínez, Albert (Institut de Recerca Biomèdica) ; Llovera, Marta (Institut de Recerca Biomèdica de LLeida) ; Wentzel, Corinna (MRC Centre for Developmental Neurobiology) ; Poopalasundaram, Subathra (MRC Centre for Developmental Neurobiology) ; Hindges, Robert (MRC Centre for Developmental Neurobiology) ; Soriano García, Eduardo (Institut de Recerca Biomèdica) ; Comella i Carnicé, Joan Xavier, 1963- (Institut de Recerca Biomèdica de LLeida) ; Drescher, Uwe (MRC Centre for Developmental Neurobiology)
Toward understanding topographically specific branching of retinal axons in their target area, we have studied the interaction between neurotrophin receptors and members of the Eph family. TrkB and its ligand BDNF are uniformly expressed in the retina and tectum, respectively, and exert a branch-promoting activity, whereas EphAs and ephrinAs are expressed in gradients in retina and tectum and can mediate a suppression of axonal branching. [...]
2008 - 10.1523/JNEUROSCI.1915-08.2008
The Journal of neuroscience, Vol. 28, No. 48 (Nov. 2008) , p. 12700-12712  
4.
10 p, 2.7 MB EndoG links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes. / Zhang, Jisheng (Institut de Recerca Biomèdica de Lleida) ; Ye, Junmei (Institut de Recerca Biomèdica de Lleida) ; Altafaj, Albert (Parc Científic de Barcelona) ; Cardona, Maria (Institut de Recerca Biomèdica de Lleida) ; Bahi, Núria (Institut de Recerca Biomèdica de Lleida) ; Llovera, Marta (Institut de Recerca Biomèdica de Lleida) ; Cañas, Xavier (Parc Científic de Barcelona) ; Cook, Stuart A. (Medical Research Council Clinical Sciences Centre (Londres, Regne Unit)) ; Comella i Carnicé, Joan Xavier, 1963- (Vall d'Hebron Hospitals) ; Sanchis, Daniel (Institut de Recerca Biomèdica de Lleida)
Mitochondrial dysfunction, caspase activation and caspase-dependent DNA fragmentation are involved in cell damage in many tissues. However, differentiated cardiomyocytes repress the expression of the canonical apoptotic pathway and their death during ischemia is caspase-independent. [...]
2011 - 10.1371/journal.pone.0017998
PLoS one, Vol. 6, Issue 3 (March 2011) , p. e17998  

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