Resultados globales: 2 registros encontrados en 0.03 segundos.
Artículos, Encontrados 2 registros
Artículos Encontrados 2 registros  
1.
40 p, 5.5 MB Cognitive Impairment Induced by Delta9-tetrahydrocannabinol Occurs through Heteromers between Cannabinoid CB and Serotonin 5-HT Receptors / Viñals, Xavier (Universitat Pompeu Fabra. Laboratori de Neurofarmacologia) ; Moreno, Estefanía (Universitat de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Lanfumey, Laurence (Université Paris Descartes. Institut de Psychiatrie et Neurosciences de Paris (UMR 894)) ; Cordomí Montoya, Arnau (Universitat Autònoma de Barcelona. Laboratori de Medicina Computacional) ; Pastor, Antoni (IMIM-Institut Hospital del Mar d'Investigacions Médiques. Integrative Pharmacology and Systems Neuroscience) ; de La Torre, Rafael (IMIM-Institut Hospital del Mar d'Investigacions Médiques. Integrative Pharmacology and Systems Neuroscience) ; Gasperini, Paola (Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas (CIBERNED)) ; Navarro, Gemma (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Howell, Lesley A. (University of East Anglia. School of Pharmacy (United Kingdom)) ; Pardo Carrasco, Leonardo (Universitat Autònoma de Barcelona. Laboratori de Medicina Computacional) ; Lluís, Carmen (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Canela, Enric I. (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; McCormick, Peter J. (University of East Anglia. School of Pharmacy (United Kingdom)) ; Maldonado, Rafael (Universitat Pompeu Fabra. Laboratori de Neurofarmacologia) ; Robledo, Patricia (IMIM-Institut Hospital del Mar d'Investigacions Médiques. Integrative Pharmacology and Systems Neuroscience)
Activation of cannabinoid CB1 receptors (CBR) by delta9-tetrahydrocannabinol (THC) produces a variety of negative effects with major consequences in cannabis users that constitute important drawbacks for the use of cannabinoids as therapeutic agents. [...]
2015 - 10.1371/journal.pbio.1002194
PLoS Biology, Vol. 13, Num. 7 (July 2015) , art. e1002194  
2.
16 p, 1020.6 KB Transgenic mice overexpressing the full-length neurotrophin receptor TrkC exhibit increased catecholaminergic neuron density in specific brain areas and increased anxiety-like behavior and panic reaction. / Dierssen, Mara (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Gratacòs, Mònica (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Sahún, Ignasi (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Martín, Miguel (Universitat Pompeu Fabra. Àrea de Neurofarmacologia) ; Gallego, Xavier (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Amador-Arjona, Alehandro (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Martínez de Lagrán María (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Murtra, Patricia (Universitat Pompeu Fabra. Àrea de Neurofarmacologia) ; Martí, Eulalia (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Pujana, Miguel A. (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Ferrer, Isidre (Hospital Prínceps d'Espanya. Àrea de neuropatologia) ; Dalfo Capella, Esther (Universitat Autònoma de Barcelona. Departament de Biologia i Bioquímica Molecular) ; Martínez-Cué, Carmen (Universidad de Cantabria. Departamento de Psicologia y Farmacologia) ; Flórez, Jesús (Universidad de Cantabria. Departamento de Psicologia y Farmacologia) ; Torres-Peraza, Jesús F. (Universitat de Barcelona. Departament de Biologia Cel·lular i Anatomia Patològica) ; Alberch i Vié, Jordi (Universitat de Barcelona. Departament de Biologia Cel·lular i Anatomia Patològica) ; Maldonado, Rafael (Universitat Pompeu Fabra. Àrea de Neurofarmacologia) ; Fillat, Cristina (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center) ; Estivill, Xavier (Biomedical Research Park. Genes and Disease Program, Genomic Regulation Center)
Accumulating evidence has suggested that neurotrophins participate in the pathophysiology of mood disorders. We have developed transgenic mice overexpressing the full-length neurotrophin-3 receptor TrkC (TgNTRK3) in the central nervous system. [...]
2006 - 10.1016/j.nbd.2006.07.015
Neurobiology of disease, Vol. 24, Num. 2 (2006) , p. 403-18  

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