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14 p, 4.2 MB STK11 (LKB1) missense somatic mutant isoforms promote tumor growth, motility and inflammation / Granado-Martínez, Paula (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Garcia-Ortega, Sara (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; González-Sánchez, Elena (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; McGrail, Kimberley (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Selgas, Rafael (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Grueso, Judit (Vall d'Hebron Institut d'Oncologia) ; Gil, Rosa (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Naldaiz-Gastesi, Neia (Present Address: Biodonostia, Neurosciences Area, Group of Neuromuscular Diseases) ; Rhodes, Ana C. (Institut d'Investigacions Biomèdiques August Pi i Sunyer) ; Hernandez-Losa, Javier (Hospital Universitari Vall d'Hebron) ; Ferrer, Berta (Hospital Universitari Vall d'Hebron) ; Canals, Francesc (Vall d'Hebron Institut d'Oncologia) ; Villanueva, Josep (Vall d'Hebron Institut d'Oncologia) ; Méndez Fernández, Olga (Vall d'Hebron Institut d'Oncologia) ; Espinosa-Gil, Sergio (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Lizcano de Vega, José Miguel (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Muñoz Couselo, Eva (Vall d'Hebron Institut d'Oncologia) ; García-Patos Briones, Vicente (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Recio Conde, Juan Ángel (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Universitat Autònoma de Barcelona
Elucidating the contribution of somatic mutations to cancer is essential for personalized medicine. STK11 (LKB1) appears to be inactivated in human cancer. However, somatic missense mutations also occur, and the role/s of these alterations to this disease remain unknown. [...]
2020 - 10.1038/s42003-020-1092-0
Communications Biology, Vol. 3 (july 2020)  
Detailed record - Creative Commons

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