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Resultats globals: 4 registres trobats en 0.03 segons.
Articles, 4 registres trobats
Articles 4 registres trobats  
1.
17 p, 2.7 MB Defective mitochondrial protein import contributes to complex I-induced mitochondrial dysfunction and neurodegeneration in Parkinson's disease / Franco Iborra, Sandra (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Cuadros, Thais (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Parent, Annabelle (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Romero-Gimenez, Jordi (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Vila Bover, Miquel (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Perier, Celine (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Universitat Autònoma de Barcelona
Mitochondria are the prime energy source in most eukaryotic cells, but these highly dynamic organelles are also involved in a multitude of cellular events. Disruption of mitochondrial homeostasis and the subsequent mitochondrial dysfunction plays a key role in the pathophysiology of Parkinson's disease (PD). [...]
2018 - 10.1038/s41419-018-1154-0
Cell death and disease, Vol. 9 (november 2018)  
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2.
18 p, 8.5 MB Identification of distinct pathological signatures induced by patient-derived α-synuclein structures in nonhuman primates / Bourdenx, M. (CNRS) ; Nioche, A. (Institut Jean Nicod, Département d'études cognitives, CNRS) ; Dovero, S. (CNRS, Institut des Maladies Neurodégénératives) ; Arotcarena, M.-L. (CNRS, Institut des Maladies Neurodégénératives) ; Camus, S. (CNRS, Institut des Maladies Neurodégénératives) ; Porras, G. (CNRS, Institut des Maladies Neurodégénératives) ; Thiolat, M.-L. (CNRS, Institut des Maladies Neurodégénératives) ; Rougier, N. P. (INRIA Bordeaux Sud-Ouest) ; Prigent, A. (CHU Nantes, Department of Neurology, Nantes) ; Aubert, P. (CHU Nantes, Department of Neurology) ; Bohic, S. (EA-7442 Rayonnement Synchrotron et Recherche Medicale, RSRM, University of Grenoble Alpes) ; Sandt, C. (SMIS beamline, Synchrotron SOLEIL, l'orme des merisiers) ; Laferrière, F. (CNRS, Institut des Maladies Neurodégénératives) ; Doudnikoff, E. (CNRS, Institut des Maladies Neurodégénératives) ; Kruse, Niels (University Medical Center Goettingen, Institute of Neuropathology) ; Mollenhauer, B. (University Medical Center Goettingen, Institute of Neuropathology) ; Novello, Salvatore (University of Ferrara. Neuroscience Center) ; Morari, M. (University of Ferrara. Neuroscience Center) ; Leste-Lasserre, T. (INSERM, Neurocentre Magendie, U1215, Physiopathologie de la Plasticité Neuronale) ; Trigo-Damas, I. (Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas) ; Goillandeau, M. (CNRS, Institut des Maladies Neurodégénératives) ; Perier, Celine (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Estrada, C. (Institute of Research on Aging (IUIE), Universidad de Murcia) ; Garcia-Carrillo, N. (Centro Experimental en Investigaciones Biomédica (CEIB), Universidad de Murcia) ; Recasens, A. (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Vaikath, N. N. (Neurological Disorders Research Center, Qatar Biomedical Research Institute (QBRI), Hamad Bin Khalifa University (HBKU), Education City, Qatar) ; El-Agnaf, O. M. A. (Neurological Disorders Research Center, Qatar Biomedical Research Institute (QBRI), Hamad Bin Khalifa University (HBKU), Education City, Qatar) ; Herrero Ezquerdo, María Trinidad (Universidad de Murcia) ; Derkinderen, Pascal (CHU Nantes, Department of Neurology) ; Vila Bover, Miquel (Institució Catalana de Recerca i Estudis Avançats) ; Obeso, J. A. (Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas) ; Dehay, B. (CNRS, Institut des Maladies Neurodégénératives) ; Bezard, E. (CNRS, Institut des Maladies Neurodégénératives) ; Universitat Autònoma de Barcelona
Machine learning-based approach unravels distinct pathological signatures induced by patient-derived α-synuclein seeds in monkeys. Dopaminergic neuronal cell death, associated with intracellular α-synuclein (α-syn)-rich protein aggregates [termed "Lewy bodies" (LBs)], is a well-established characteristic of Parkinson's disease (PD). [...]
2020 - 10.1126/sciadv.aaz9165
Science advances, Vol. 6 (may 2020)  
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3.
25 p, 2.6 MB Mitochondrial Quality Control in Neurodegenerative Diseases : Focus on Parkinson's Disease and Huntington's Disease / Franco Iborra, Sandra (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Vila Bover, Miquel (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular) ; Perier, Celine (Hospital Universitari Vall d'Hebron. Institut de Recerca)
In recent years, several important advances have been made in our understanding of the pathways that lead to cell dysfunction and death in Parkinson's disease (PD) and Huntington's disease (HD). Despite distinct clinical and pathological features, these two neurodegenerative diseases share critical processes, such as the presence of misfolded and/or aggregated proteins, oxidative stress, and mitochondrial anomalies. [...]
2018 - 10.3389/fnins.2018.00342
Frontiers in Neuroscience, Vol. 12 (May 2018) , art. 342  
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4.
10 p, 2.8 MB DYRK1A promotes dopaminergic neuron survival in the developing brain and in a mouse model of Parkinson's disease / Barallobre, M. J. (Institut de Biologia Molecular de Barcelona) ; Perier, Celine (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Bové, Jordi (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Laguna, Ariadna (Institut de Biologia Molecular de Barcelona) ; Delabar, J. M. (Université Paris Diderot. Unité de Biologie Fonctionnelle et Adaptative) ; Vila Bover, Miquel (Institució Catalana de Recerca i Estudis Avançats) ; Arbonés, M. L. (Institut de Biologia Molecular de Barcelona) ; Universitat Autònoma de Barcelona
In the brain, programmed cell death (PCD) serves to adjust the numbers of the different types of neurons during development, and its pathological reactivation in the adult leads to neurodegeneration. [...]
2014 - 10.1038/cddis.2014.253
Cell death and disease, Vol. 5 (06 2014) , p. e1289  
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