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Articles, 2 records found
Articles 2 records found  
1.
16 p, 1.8 MB Chemotherapeutic agent 5-fluorouracil increases survival of SOD1 mouse model of ALS / Rando, Amaya (Instituto de Investigación Sanitaria Aragón. Centro de Investigación Biomédica de Aragón (CIBA)) ; De La Torre, M. (Instituto de Investigación Sanitaria Aragón. Centro de Investigación Biomédica de Aragón (CIBA)) ; Martinez-Muriana, Anna (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Zaragoza, P. (Instituto de Investigación Sanitaria Aragón. Centro de Investigación Biomédica de Aragón (CIBA)) ; Musaro, A. (Center for Life Nano Science@Sapienza. Istituto Italiano di Tecnologia) ; Hernández, S. (Departament de Medicina Experimental. Grup Patologia Neuromuscular Experimental. Facultat de Medicina. Universitat de Lleida/IRBLLEIDA) ; Navarro, Xavier (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Toivonen, J.M. (Instituto de Investigación Sanitaria Aragón. Centro de Investigación Biomédica de Aragón (CIBA)) ; Osta, R. (Instituto de Investigación Sanitaria Aragón. Centro de Investigación Biomédica de Aragón (CIBA))
Amyotrophic lateral sclerosis (ALS) is a lethal motor neuron disease with no cure. Currently there are only two ALS drugs approved by the FDA, both with a limited therapeutic effect. In the search for drug candidates for ALS, we studied the effect of known stem cell mobilizing agents (treatment) and antimetabolite 5-fluorouracil (5-FU) (anti-treatment) in SOD1G93A model of ALS. [...]
2019 - 10.1371/journal.pone.0210752
PloS one, Vol. 14 Núm. 1 (january 2019) , p. e0210752  
2.
10 p, 3.4 MB Neuroprotective Effect of Non-viral Gene Therapy Treatment Based on Tetanus Toxin C-fragment in a Severe Mouse Model of Spinal Muscular Atrophy / Oliván, Sara (Universidad de Zaragoza. Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomedicina. Grupo AMB.) ; Calvo, Ana C. (Universidad de Zaragoza. Facultad de Veterinaria. Laboratorio de Genética Bioquímica) ; Rando, Amaya (Universidad de Zaragoza. Facultad de Veterinaria. Laboratorio de Genética Bioquímica) ; Herrando-Grabulosa, Mireia (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Manzano, Raquel (University of Oxford. Department of Physiology, Anatomy and Genetics) ; Zaragoza, Pilar (Universidad de Zaragoza. Instituto Agroalimentario de Aragón (IA2)) ; Tizzano, Eduardo F. (Hospital Vall d'Hebron. Àrea de Genètica Clínica i Molecular. Centros de Investigación Biomédica en Red) ; Aguilera Ávila, José (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Osta, Rosario (Universidad de Zaragoza. Centro de Investigación y Tecnología Agroalimentaria de Aragón) ; Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular
Spinal muscular atrophy (SMA) is a hereditary childhood disease that causes paralysis and progressive degeneration of skeletal muscles and spinal motor neurons. SMA is associated with reduced levels of full-length Survival of Motor Neuron (SMN) protein, due to mutations in the Survival of Motor Neuron 1 gene. [...]
2016 - 10.3389/fnmol.2016.00076
Frontiers in molecular neuroscience, Vol. 9 (August 2016) , art. 76  

See also: similar author names
1 Rando, A.
2 Rando, Ariadna
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