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Articles, 2 records found
Articles 2 records found  
1.
11 p, 1.0 MB Cardiomyocyte hypertrophy induced by Endonuclease G deficiency requires reactive oxygen radicals accumulation and is inhibitable by the micropeptide humanin / Blasco, Natividad (Institut de Recerca Biomèdica de Lleida) ; Cámara, Yolanda (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Núñez, Estefanía (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares) ; Beà, Aida (Institut de Recerca Biomèdica de Lleida) ; Barés, Gisel (Institut de Recerca Biomèdica de Lleida) ; Forné, Carles (Institut de Recerca Biomèdica de Lleida) ; Ruíz-Meana, Marisol (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Girón, Cristina (Institut de Recerca Biomèdica de Lleida) ; Barba, Ignasi (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Garcia-Arumi, Elena (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; García-Dorado, David (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Vázquez, Jesús (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares) ; Martí, Ramon A. (Hospital Universitari Vall d'Hebron. Institut de Recerca) ; Llovera, Marta (Institut de Recerca Biomèdica de Lleida) ; Sanchis, Daniel (Institut de Recerca Biomèdica de Lleida)
The endonuclease G gene (Endog), which codes for a mitochondrial nuclease, was identified as a determinant of cardiac hypertrophy. How ENDOG controls cardiomyocyte growth is still unknown. Thus, we aimed at finding the link between ENDOG activity and cardiomyocyte growth. [...]
2018 - 10.1016/j.redox.2018.02.021
Redox biology, Vol. 16 (march 2018) , p. 146-156  
2.
10 p, 2.7 MB EndoG links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes / Zhang, Jisheng (Institut de Recerca Biomèdica de Lleida) ; Ye, Junmei (Institut de Recerca Biomèdica) ; Altafaj, Albert (Parc Científic de Barcelona) ; Cardona, Maria (Institut de Recerca Biomèdica) ; Bahi, Núria (Institut de Recerca Biomèdica) ; Llovera, Marta (Institut de Recerca Biomèdica) ; Cañas, Xavier (Parc Científic de Barcelona) ; Cook, Stuart A. (Medical Research Council Clinical Sciences Centre (Londres, Regne Unit)) ; Comella i Carnicé, Joan Xavier, 1963- (Hospital Universitari Vall d'Hebron) ; Sanchis, Daniel (Institut de Recerca Biomèdica)
Mitochondrial dysfunction, caspase activation and caspase-dependent DNA fragmentation are involved in cell damage in many tissues. However, differentiated cardiomyocytes repress the expression of the canonical apoptotic pathway and their death during ischemia is caspase-independent. [...]
2011 - 10.1371/journal.pone.0017998
PloS one, Vol. 6, Issue 3 (March 2011) , p. e17998  

See also: similar author names
1 Sanchis, D.
2 Sanchis, D..
1 Sanchís, D.
1 Sanchís, D.B.
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