UAB Digital Repository of Documents 6 records found  Search took 0.02 seconds. 
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2 p, 165.1 KB Editorial - Glial Cells : Managers of Neuro-Immunity / Barcia González, Carlos (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Guillemin, Gilles J. (Macquarie University. Department of Biomedical Sciences) ; Curtin, James F. (Dublin Institute of Technology. School of Food Science & Environmental Health) ; Zirger, Jeffrey M. (U.S. Centers for Disease Control and Prevention) ; Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular
After many decades of study in the field of Neuroscience that were mostly centered on the neuron there is a mounting interest in the study of the function of the glial cells in many aspects and functions of the central nervous system. [...]
2016 - 10.3389/fncel.2016.00060
Frontiers in Cellular Neuroscience, Vol. 10 (March 2016) , art.60  
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10 p, 1.8 MB Choroid plexus dysfunction impairs beta-amyloid clearance in a triple transgenic mouse model of Alzheimer's disease / González-Marrero, Ibrahim (Universidad de La Laguna. Departamento de Anatomía Humana) ; Giménez Llort, Lydia (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Johanson, Conrad E. (Alpert Medical School at Brown University. Department of Neurosurgery) ; Carmona-Calero, Emilia María (Universidad de La Laguna. Departamento de Anatomía Humana) ; Castañeyra-Ruiz, Leandro (Universidad de La Laguna. Departamento de Anatomía Humana) ; Brito-Armas, José Miguel (Universidad de La Laguna. Departamento de Psicología) ; Castañeyra-Perdomo, Agustín (Universidad de La Laguna. Departamento de Anatomía Humana) ; Castro-Fuentes, Rafael (Universidad de La Laguna. Departamento de Psicología) ; Universitat Autònoma de Barcelona. Departament de Psiquiatria i de Medicina Legal
Compromised secretory function of choroid plexus (CP) and defective cerebrospinal fluid (CSF) production, along with accumulation of beta-amyloid (Aβ) peptides at the blood-CSF barrier (BCSFB), contribute to complications of Alzheimer's disease (AD). [...]
2015 - 10.3389/fncel.2015.00017
Frontiers in Cellular Neuroscience, Vol. 9 (February 2015) , art. 17  
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12 p, 418.6 KB Are microglial cells the regulators of lymphocyte responses in the CNS? / Almolda Ardid, Beatriz (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia) ; González, Berta (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia) ; Castellano López, Bernardo (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia)
The infiltration of immune cells in the central nervous system is a common hallmark in different neuroinflammatory conditions. Accumulating evidence indicates that resident glial cells can establish a cross-talk with infiltrated immune cells, including T-cells, regulating their recruitment, activation and function within the CNS. [...]
2015 - 10.3389/fncel.2015.00440
Frontiers in cellular neuroscience, Vol. 16 Núm. 9 (November 2015) , Article ID 440  
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14 p, 2.8 MB Gene expression parallels synaptic excitability and plasticity changes in Alzheimer’s disease / Saura Antolín, Carlos A. (Carlos Alberto) (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Parra Damas, Arnaldo J. (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Enríquez-Barreto, Lilian (Universitat Autònoma de Barcelona. Institut de Neurociències)
Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by abnormal accumulation of β-amyloid and tau and synapse dysfunction in memory-related neural circuits. Pathological and functional changes in the medial temporal lobe, a region essential for explicit memory encoding, contribute to cognitive decline in AD. [...]
2015 - 10.3389/fncel.2015.00318
Frontiers in Cellular Neuroscience, Vol. 9 Núm. 318 (2015)  
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10 p, 2.3 MB Imaging the microanatomy of astrocyte–T-cell interactions in immune-mediated inflammation / Barcia Mariño, Carlos (Consorci Hospital General Universitari (Valencia). Departament de Neurocirurgia) ; Mitxitorena, Izaskun (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Carrillo-de Sauvage, Maria A. (Institut d’Imagerie Biomédicale) ; Gallego, José Maria (Consorci Hospital General Universitari (Valencia). Departament de Neurocirurgia) ; Pérez-Vallés, Ana (Consorci Hospital General Universitari (Valencia). Departament de Patologia) ; Barcia González, Carlos (Universitat Autònoma de Barcelona. Institut de Neurociències)
The role of astrocytes in the immune-mediated inflammatory response in the brain is more prominent than previously thought. Astrocytes become reactive in response to neuro-inflammatory stimuli through multiple pathways, contributing significantly to the machinery that modifies the parenchymal environment. [...]
2013 - 10.3389/fncel.2013.00058
Frontiers in cellular neuroscience, Vol. 7 (April 2013) , p. 1-10  
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11 p, 3.3 MB Differential effects on KCC2 expression and spasticity of ALS and traumatic injuries to motoneurons / Mòdol, Laura (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Mancuso, Renzo (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Alé, Albert (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Francos Quijorna, Isaac (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Navarro, X. (Xavier) (Universitat Autònoma de Barcelona. Departament de Biologia Cel·lular, de Fisiologia i d'Immunologia)
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease manifested by progressive muscle atrophy and paralysis due to the loss of upper and lower motoneurons (MN). Spasticity appears in ALS patients leading to further disabling consequences. [...]
2014 - 10.3389/fncel.2014.00007
Frontiers in cellular neuroscience, Vol. 8 Núm. 7 (Jan. 2014) , p. 1-11  

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