Dipòsit Digital de Documents de la UAB 32 registres trobats  1 - 10següentfinal  anar al registre: La cerca s'ha fet en 0.01 segons. 
1.
15 p, 1.4 MB Corticomotor Plasticity Predicts Clinical Efficacy of Combined Neuromodulation and Cognitive Training in Alzheimer's Disease / Brem, Anna Katharine (Beth Israel Deaconess Medical Center) ; Di Iorio, Riccardo (Polyclinic A. Gemelli Foundation - IRCCS, Rome) ; Fried, Peter J. (Beth Israel Deaconess Medical Center) ; Oliveira Maia, Albino J. (Champalimaud Centre for the Unknown, Lisbon) ; Marra, Camillo (Polyclinic A. Gemelli Foundation - IRCCS, Rome) ; Profice, Paolo (Polyclinic A. Gemelli Foundation - IRCCS, Rome) ; Quaranta, Davide (Polyclinic A. Gemelli Foundation - IRCCS, Rome) ; Schilberg, Lukas (Beth Israel Deaconess Medical Center) ; Atkinson, Natasha J. (Beth Israel Deaconess Medical Center) ; Seligson, Erica E. (Beth Israel Deaconess Medical Center) ; Rossini, Paolo Maria (IRCCS San Raffaele, Rome) ; Pascual Leone, Alvaro (Institut Germans Trias i Pujol. Institut Guttmann) ; Universitat Autònoma de Barcelona
To investigate the efficacy of repetitive transcranial magnetic stimulation (rTMS) combined with cognitive training for treatment of cognitive symptoms in patients with Alzheimer's disease (AD). A secondary objective was to analyze associations between brain plasticity and cognitive effects of treatment. [...]
2020 - 10.3389/fnagi.2020.00200
Frontiers in aging neuroscience, Vol. 12 (july 2020)  
2.
15 p, 1.2 MB Plasma Protein Biomarkers for the Prediction of CSF Amyloid and Tau and [ 18 F]-Flutemetamol PET Scan Result / Westwood, Sarah (University of Oxford. Department of Psychiatry) ; Baird, Alison L. (Department of Psychiatry, University of Oxford) ; Hye, Abdul (Biomedical Research Unit for Dementia, NIHR Biomedical Research Centre for Mental Health, South London and Maudsley NHS Foundation Trust) ; Ashton, Nicholas J. (Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg) ; Nevado-Holgado, Alejo J. (Department of Psychiatry, University of Oxford) ; Anand, Sneha N. (Department of Psychiatry, University of Oxford) ; Liu, Benjamine (Department of Psychiatry, University of Oxford) ; Newby, Danielle (Department of Psychiatry, University of Oxford) ; Bazenet, Chantal (Maurice Wohl Clinical Neuroscience Institute, Institute of Psychiatry, Psychology and Neuroscience, King's College London) ; Kiddle, Steven J. (MRC Biostatistics Unit, Cambridge Biomedical Campus, Cambridge Institute of Public Health, University of Cambridge) ; Ward, Malcolm (Proteomics Facility, Institute of Psychiatry, Psychology & Neuroscience, King's College London) ; Newton, Ben (GE Healthcare Life Sciences Core Imaging) ; Desai, Keyur (Biosciences, GE Global Research) ; Tan Hehir, Cristina (Biosciences, GE Global Research) ; Zanette, Michelle (GE Healthcare Life Sciences Core Imaging) ; Galimberti, Daniela (Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico) ; Parnetti, Lucilla (Center for Memory Disorders and Laboratory of Clinical Neurochemistry, Neurology Clinic, University of Perugia) ; Lleó, Alberto (Institut d'Investigació Biomèdica Sant Pau) ; Baker, Susan (Janssen Neuroscience Research & Development) ; Narayan, Vaibhav A. (Janssen Neuroscience Research & Development) ; van der Flier, Wiesje M. (Department of Epidemiology and Biostatistics, VU University Medical Center) ; Scheltens, Philip (Department of Neurology, Alzheimer Centre, Amsterdam Neuroscience, VU University Medical Center) ; Teunissen, Charlotte E. (Department of Clinical Chemistry, Neurochemistry Lab and Biobank, Amsterdam Neuroscience, VU University Medical Center) ; Visser, Pieter Jelle (Department of Psychiatry and Neuropsychology, School for Mental Health and Neuroscience, Maastricht University) ; Lovestone, Simon (Department of Psychiatry, University of Oxford) ; Universitat Autònoma de Barcelona
Background: Blood biomarkers may aid in recruitment to clinical trials of Alzheimer's disease (AD) modifying therapeutics by triaging potential trials participants for amyloid positron emission tomography (PET) or cerebrospinal fluid (CSF) Aβ and tau tests. [...]
2018 - 10.3389/fnagi.2018.00409
Frontiers in aging neuroscience, Vol. 10 (december 2018)  
3.
12 p, 3.6 MB ZPD-2, a small compound that inhibits α-synuclein amyloid aggregation and its seeded polymerization / Peña Díaz, Samuel (Institut de Biotecnologia i de Biomedicina "Vicent Villar Palasí") ; Pujols, Jordi (Institut de Biotecnologia i de Biomedicina "Vicent Villar Palasí") ; Conde Giménez, María (Universidad de Zaragoza. Instituto Universitario de Investigación de Biocomputación y Física de Sistemas Complejos) ; Čarija, Anita (Institut de Biotecnologia i de Biomedicina "Vicent Villar Palasí") ; Dalfo, Esther (Universitat Autònoma de Barcelona. Facultat de Medicina) ; García, Jesús (Institut de Recerca Biomèdica) ; Navarro Cantero, Susanna (Institut de Biotecnologia i de Biomedicina "Vicent Villar Palasí") ; Pinheiro, Francisca (Institut de Biotecnologia i de Biomedicina "Vicent Villar Palasí") ; Santos Suárez, Jaime (Universitat Autonoma de Barcelona. Departament de Bioquimica i Biologia Molecular) ; Salvatella, Xavier (Institut de Recerca Biomèdica) ; Sancho, Javier (Universidad de Zaragoza. Instituto Universitario de Investigación de Biocomputación y Física de Sistemas Complejos) ; Ventura, Salvador (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular)
α-Synuclein (α-Syn) forms toxic intracellular protein inclusions and transmissible amyloid structures in Parkinson's disease (PD). Preventing α-Syn self-assembly has become one of the most promising approaches in the search for disease-modifying treatments for this neurodegenerative disorder. [...]
2019 - 10.3389/fnmol.2019.00306
Frontiers in molecular neuroscience, Vol. 12 (Dec. 2019) , art. 306  
4.
15 p, 1.2 MB GRP78 at the centre of the stage in cancer and neuroprotection / Casas Louzao, Caty (Universitat Autònoma de Barcelona. Institut de Neurociències)
The 78-kDa glucose-regulated protein GRP78, also known as BiP and HSP5a, is a multifunctional protein with activities far beyond its well-known role in the unfolded protein response (UPR) which is activated after endoplasmic reticulum (ER) stress in the cells. [...]
2017 - 10.3389/fnins.2017.00177
Frontiers in Neuroscience, Vol. 11, (May 2017) , p. 177  
5.
25 p, 2.6 MB Mitochondrial Quality Control in Neurodegenerative Diseases : Focus on Parkinson's Disease and Huntington's Disease / Franco-Iborra, Sandra (Vall d'Hebron. Institut de Recerca (VHIR)) ; Vila Bover, Miquel (Universitat Autònoma de Barcelona. Departament de Bioquímica i Biologia Molecular) ; Perier, Celine (Vall d'Hebron. Institut de Recerca (VHIR))
In recent years, several important advances have been made in our understanding of the pathways that lead to cell dysfunction and death in Parkinson's disease (PD) and Huntington's disease (HD). Despite distinct clinical and pathological features, these two neurodegenerative diseases share critical processes, such as the presence of misfolded and/or aggregated proteins, oxidative stress, and mitochondrial anomalies. [...]
2018 - 10.3389/fnins.2018.00342
Frontiers in Neuroscience, Vol. 12 (May 2018) , art. 342  
6.
11 p, 4.2 MB Neuregulin 1 Reduces Motoneuron Cell Death and Promotes Neurite Growth in an in Vitro Model of Motoneuron Degeneration / Mòdol Caballero, Guillem (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Santos, Daniel (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Navarro, Xavier (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Herrando-Grabulosa, Mireia (Universitat Autònoma de Barcelona. Institut de Neurociències)
Amyotrophic Lateral Sclerosis (ALS) is a devastating neurodegenerative disorder with no effective treatment currently available. Although the mechanisms of motoneuron (MN) death are still unclear, glutamate excitotoxicity and neuroinflammatory reaction are two main features in the neurodegenerative process of ALS. [...]
2018 - 10.3389/fncel.2017.00431
Frontiers in cellular neuroscience, Vol. 11 (january 2018)  
7.
14 p, 3.4 MB Slow and Fast Neocortical Oscillations in the Senescence-Accelerated Mouse Model SAMP8 / Castano-Prat, Patricia (Systems Neuroscience, Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)) ; Perez-Zabalza, Maria (Systems Neuroscience, Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)) ; Perez-Mendez, Lorena (Systems Neuroscience, Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS)) ; Escorihuela, Rosa M. (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Sanchez-Vives, Maria V. (ICREA)
The senescence-accelerated mouse prone 8 (SAMP8) model is characterized by accelerated, progressive cognitive decline as well as Alzheimer's disease (AD)-like neurodegenerative changes, and resembles the etiology of multicausal, sporadic late-onset/age-related AD in humans. [...]
2017 - 10.3389/fnagi.2017.00141
Frontiers in aging neuroscience, Vol. 9 (may 2017)  
8.
13 p, 1.6 MB Neuronal Mitophagy in Neurodegenerative Diseases / Martinez-Vicente, Marta (Hospital Universitari Vall d'Hebron. Institut de Recerca)
Neuronal homeostasis depends on the proper functioning of different quality control systems. All intracellular components are subjected to continuous turnover through the coordinated synthesis, degradation and recycling of their constituent elements. [...]
2017 - 10.3389/fnmol.2017.00064
Frontiers in molecular neuroscience, Vol. 10 (march 2017)  
9.
9 p, 894.2 KB VNTR-DAT1 and COMTVal158Met Genotypes Modulate Mental Flexibility and Adaptive Behavior Skills in Down Syndrome / del Hoyo, Laura (Universitat Autònoma de Barcelona. Departament de Farmacologia, de Terapèutica i de Toxicologia) ; Xicota, Laura (Institut Hospital del Mar d'Investigacions Mèdiques) ; Langohr, Klaus (Institut Hospital del Mar d'Investigacions Mèdiques) ; Sánchez-Benavides, Gonzalo (Institut Hospital del Mar d'Investigacions Mèdiques) ; de Sola, Susana (Institut Hospital del Mar d'Investigacions Mèdiques) ; Cuenca-Royo, Aida (Institut Hospital del Mar d'Investigacions Mèdiques) ; Rodriguez, Joan (Institut Hospital del Mar d'Investigacions Mèdiques) ; Rodríguez-Morató, Jose (Institut Hospital del Mar d'Investigacions Mèdiques) ; Farré Albaladejo, Magí (Universitat Autònoma de Barcelona. Departament de Farmacologia, de Terapèutica i de Toxicologia) ; Dierssen, Mara (CIBER de Enfermedades Raras, Instituto Salud Carlos III) ; de la Torre, Rafael (CInstitut Hospital del Mar d'Investigacions Mèdiques)
Down syndrome (DS) is an aneuploidy syndrome that is caused by trisomy for human chromosome 21 resulting in a characteristic cognitive and behavioral phenotype, which includes executive functioning and adaptive behavior difficulties possibly due to prefrontal cortex (PFC) deficits. [...]
2016 - 10.3389/fnbeh.2016.00193
Frontiers in Behavioral Neuroscience, Vol. 10 (october 2016)  
10.
10 p, 3.4 MB Neuroprotective Effect of Non-viral Gene Therapy Treatment Based on Tetanus Toxin C-fragment in a Severe Mouse Model of Spinal Muscular Atrophy / Oliván, Sara (Universidad de Zaragoza. Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomedicina. Grupo AMB.) ; Calvo, Ana C. (Universidad de Zaragoza. Facultad de Veterinaria. Laboratorio de Genética Bioquímica) ; Rando, Amaya (Universidad de Zaragoza. Facultad de Veterinaria. Laboratorio de Genética Bioquímica) ; Herrando-Grabulosa, Mireia (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Manzano, Raquel (University of Oxford. Department of Physiology, Anatomy and Genetics) ; Zaragoza, Pilar (Universidad de Zaragoza. Instituto Agroalimentario de Aragón (IA2)) ; Tizzano, Eduardo F. (Hospital Vall d'Hebron. Àrea de Genètica Clínica i Molecular. Centros de Investigación Biomédica en Red) ; Aguilera Ávila, José (Universitat Autònoma de Barcelona. Institut de Neurociències) ; Osta, Rosario (Universidad de Zaragoza. Centro de Investigación y Tecnología Agroalimentaria de Aragón) ; Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular
Spinal muscular atrophy (SMA) is a hereditary childhood disease that causes paralysis and progressive degeneration of skeletal muscles and spinal motor neurons. SMA is associated with reduced levels of full-length Survival of Motor Neuron (SMN) protein, due to mutations in the Survival of Motor Neuron 1 gene. [...]
2016 - 10.3389/fnmol.2016.00076
Frontiers in molecular neuroscience, Vol. 9 (August 2016) , art. 76  

Dipòsit Digital de Documents de la UAB : 32 registres trobats   1 - 10següentfinal  anar al registre:
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