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18 p, 7.7 MB Type I IFN exacerbates disease in tuberculosis-susceptible mice by inducing neutrophil-mediated lung inflammation and NETosis / Moreira-Teixeira, Lúcia (The Francis Crick Institute, London) ; Stimpson, Philippa J. (The Francis Crick Institute, London) ; Stavropoulos, Evangelos (The Francis Crick Institute, London) ; Hadebe, Sabelo (The Francis Crick Institute, London) ; Chakravarty, Probir (The Francis Crick Institute, London) ; Ioannou, Marianna (The Francis Crick Institute, London) ; Aramburu, Iker Valle (The Francis Crick Institute, London) ; Herbert, Eleanor (The Francis Crick Institute, London) ; Priestnall, Simon L. (The Francis Crick Institute, London) ; Suarez-Bonnet, Alejandro (The Francis Crick Institute, London) ; Sousa, Jeremy (Universidade do Porto. ICBAS - Instituto de Ciências Biomédicas Abel Salazar, Porto) ; Fonseca, Kaori L. (Instituto Gulbenkian de Ciência (IGC), Oeiras) ; Wang, Qian (The Francis Crick Institute, London) ; Vashakidze, Sergo (National Center for Tuberculosis and Lung Diseases (NCTLD), Tbilisi) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vilaplana, Cristina (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Saraiva, Margarida (Universidade do Porto. IBMC - Instituto de Biologia Molecular e Celular) ; Papayannopoulos, Venizelos (The Francis Crick Institute, London) ; O'Garra, Anne (Imperial College London)
Tuberculosis (TB) is a leading cause of mortality due to infectious disease, but the factors determining disease progression are unclear. Transcriptional signatures associated with type I IFN signalling and neutrophilic inflammation were shown to correlate with disease severity in mouse models of TB. [...]
2020 - 10.1038/s41467-020-19412-6
Nature communications, Vol. 11 (november 2020)  
Registre complet - Creative Commons

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