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16 p, 4.3 MB Ischemic postconditioning reduces reperfusion arrhythmias by adenosine receptors and protein kinase C activation but is independent of KATP channels or connexin 43 / Diez, E. R. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Sánchez, J. A. (Universitat Autònoma de Barcelona. Departament de Medicina) ; Prado, N. J. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Zumino, A. Z. P. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; García-Dorado, David (Universitat Autònoma de Barcelona. Departament de Medicina) ; Miatello, R. M. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Rodríguez-Sinovas, A. (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares)
Ischemic postconditioning (IPoC) reduces reperfusion arrhythmias but the antiarrhythmic mechanisms remain unknown. The aim of this study was to analyze IPoC electrophysiological effects and the role played by adenosine A, A and A receptors, protein kinase C, ATP-dependent potassium (K) channels, and connexin 43. [...]
2019 - 10.3390/ijms20235927
International journal of molecular sciences, Vol. 20 Núm. 23 (january 2019) , p. 5927  

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