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Resultados globales: 5 registros encontrados en 0.02 segundos.
Artículos, Encontrados 5 registros
Artículos Encontrados 5 registros  
1.
9 p, 1.1 MB Conventional and digital Ki67 evaluation and their correlation with molecular prognosis and morphological parameters in luminal breast cancer / Pons, Laura (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Hernández-León, Laura (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Altaleb, Ahmad (Mubarak Al-Kabeer Hospital, Jabriya, Kuwait) ; Ussene, Esperança (Hospital Vila Franca de Xira, Portugal) ; Iglesias, Roman (Hospital Santa Barbara, Ciudad Real) ; Castillo, Ana (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Castellà Fernández, Eva (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Quiroga, Vanesa (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Felip, Eudald (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Cirauqui, Beatriz (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Margelí, Mireia (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Fernández, Pedro Luis (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Universitat Autònoma de Barcelona
Digital counting methods were developed to decrease the high intra- and inter-observer variability of immunohistochemical markers such as Ki67, with most presenting a good correlation coefficient (CC). [...]
2022 - 10.1038/s41598-022-11411-5
Scientific reports, Vol. 12 (may 2022)  
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2.
9 p, 1.2 MB Results of a worldwide survey on the currently used histopathological diagnostic criteria for invasive lobular breast cancer / De Schepper, Maxim (KU Leuven) ; Vincent-Salomon, Anne (PSL Research University) ; Christgen, Matthias (Hannover Medical School) ; Van Baelen, Karen (KU Leuven) ; Richard, François (KU Leuven) ; Tsuda, Hitoshi (National Defense Medical College Hospital) ; Kurozumi, Sasagu (International University of Health and Welfare. Department of Breast Surgery) ; Brito, Maria Jose (Champalimaud Foundation (Portugal)) ; Cserni, Gabor (University of Szeged) ; Schnitt, Stuart (Brigham and Women's Hospital (Boston, Estats Units d'Amèrica)) ; Larsimont, Denis (Université Libre de Bruxelles. Department of Pathology) ; Kulka, Janina (Semmelweis University. Department of Pathology, Forensic and Insurance Medicine) ; Fernández, Pedro Luis (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Olivar, Ana Aula (Hospital Universitari de Girona Doctor Josep Trueta) ; Melendez, Cristina (Hospital Universitari de Girona Doctor Josep Trueta) ; Van Bockstal, Mieke (Cliniques universitaires Saint-Luc Bruxelles) ; Kovacs, Aniko (Sahlgrenska University Hospital. Department of Clinical Pathology) ; Varga, Zsuzsanna (University Hospital Zurich (Suïssa)) ; Wesseling, Jelle (The Netherlands Cancer Institute (Amsterdam, Països Baixos)) ; Bhargava, Rohit (UPMC Magee-Womens Hospital. Department of Pathology) ; Boström, Pia (University of Turku. Department of Pathology) ; Franchet, Camille (Institut Universitaire du Cancer Toulouse-Oncopole) ; Zambuko, Blessing (University of Botswana. Department of Pathology) ; Matute, Gustavo (Universidad Pontificia Bolivariana) ; Mueller, Sophie (Hannover Medical School) ; Berghian, Anca (Centre Henri Becquerel. Department of Biopathology) ; Rakha, Emad (Nottingham University Hospitals NHS Trust (Regne Unit)) ; van Diest, Paul J. (University Medical Center Utrecht. Department of Pathology) ; Oesterreich, Steffi (Magee-Womens Research Institute (Estats Units d'Amèrica)) ; Derksen, Patrick W. B. (University Medical Center Utrecht. Department of Pathology) ; Floris, Giuseppe (University Hospitals Leuven (Bèlgica)) ; Desmedt, Christine (KU Leuven) ; Universitat Autònoma de Barcelona
Invasive lobular carcinoma (ILC) represents the second most common subtype of breast cancer (BC), accounting for up to 15% of all invasive BC. Loss of cell adhesion due to functional inactivation of E-cadherin is the hallmark of ILC. [...]
2022 - 10.1038/s41379-022-01135-2
Modern Pathology, Vol. 35 (august 2022) , p. 1812-1820  
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3.
12 p, 3.1 MB A Beneficial Effect of Low-Dose Aspirin in a Murine Model of Active Tuberculosis / Kroesen, Vera Marie (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; García, Eric (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rosales, Yaiza (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Díaz, Jorge (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Martín-Céspedes, Montse (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Tapia, Gustavo (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Sarrias, Maria-Rosa (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Cardona, Pere-Joan (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vilaplana, Cristina (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol)
An excessive, non-productive host-immune response is detrimental in active, chronic tuberculosis (TB) disease as it typically leads to tissue damage. Given their anti-inflammatory effect, non-steroidal anti-inflammatory drugs can potentially attenuate excessive inflammation in active TB disease. [...]
2018 - 10.3389/fimmu.2018.00798
Frontiers in immunology, Vol. 9 (april 2018)  
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4.
18 p, 7.7 MB Type I IFN exacerbates disease in tuberculosis-susceptible mice by inducing neutrophil-mediated lung inflammation and NETosis / Moreira-Teixeira, Lúcia (The Francis Crick Institute, London) ; Stimpson, Philippa J. (The Francis Crick Institute, London) ; Stavropoulos, Evangelos (The Francis Crick Institute, London) ; Hadebe, Sabelo (The Francis Crick Institute, London) ; Chakravarty, Probir (The Francis Crick Institute, London) ; Ioannou, Marianna (The Francis Crick Institute, London) ; Aramburu, Iker Valle (The Francis Crick Institute, London) ; Herbert, Eleanor (The Francis Crick Institute, London) ; Priestnall, Simon L. (The Francis Crick Institute, London) ; Suarez-Bonnet, Alejandro (The Francis Crick Institute, London) ; Sousa, Jeremy (Universidade do Porto. ICBAS - Instituto de Ciências Biomédicas Abel Salazar, Porto) ; Fonseca, Kaori L. (Instituto Gulbenkian de Ciência (IGC), Oeiras) ; Wang, Qian (The Francis Crick Institute, London) ; Vashakidze, Sergo (National Center for Tuberculosis and Lung Diseases (NCTLD), Tbilisi) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vilaplana, Cristina (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Saraiva, Margarida (Universidade do Porto. IBMC - Instituto de Biologia Molecular e Celular) ; Papayannopoulos, Venizelos (The Francis Crick Institute, London) ; O'Garra, Anne (Imperial College London)
Tuberculosis (TB) is a leading cause of mortality due to infectious disease, but the factors determining disease progression are unclear. Transcriptional signatures associated with type I IFN signalling and neutrophilic inflammation were shown to correlate with disease severity in mouse models of TB. [...]
2020 - 10.1038/s41467-020-19412-6
Nature communications, Vol. 11 (november 2020)  
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5.
13 p, 3.5 MB Deficiency in the glycosyltransferase Gcnt1 increases susceptibility to tuberculosis through a mechanism involving neutrophils / Fonseca, Kaori L. (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Maceiras, Ana Raquel (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Matos, Rita (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Simoes-Costa, Luisa (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Sousa, Jeremy (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Cá, Baltazar (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Barros, Leandro (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Fernandes, Ana Isabel (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Mereiter, Stefan (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Reis, Ricardo (CDP-Centro de Diagnóstico Pneumológico do Porto) ; Gomes, Joana (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Tapia, Gustavo (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Rodríguez-Martínez, Paula (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Martín-Céspedes, Montse (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Vashakidze, Sergo (National Center for Tuberculosis and Lung Diseases (NCTLD). Tbilisi) ; Gogishvili, Shota (National Center for Tuberculosis and Lung Diseases (NCTLD). Tbilisi) ; Nikolaishvili, Keti (National Center for Tuberculosis and Lung Diseases (NCTLD). Tbilisi) ; Appelberg, Rui (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Gärtner, Fátima (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Rodrigues, Pedro N. S. (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Vilaplana, Cristina (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Reis, Celso A. (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Magalhães, Ana (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Saraiva, Margarida (Universidade do Porto. Instituto de Investigação e Inovação em Saúde) ; Universitat Autònoma de Barcelona
Modulation of immunity and disease by glycans is increasingly recognized. However, how host glycosylation shapes and is shaped by tuberculosis remains poorly understood. We show that deficiency in the glucosaminyl (N-acetyl) transferase 1 (Gcnt1), a key enzyme for core-2 O -glycans biosynthesis, drives susceptibility to Mycobacterium tuberculosis infection. [...]
2020 - 10.1038/s41385-020-0277-7
Mucosal Immunology, Vol. 13 (march 2020) , p. 836-848  
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