Ischemic postconditioning reduces reperfusion arrhythmias by adenosine receptors and protein kinase C activation but is independent of KATP channels or connexin 43 - Diez, E. R. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Sánchez, J. A. (Universitat Autònoma de Barcelona. Departament de Medicina) ; Prado, N. J. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Zumino, A. Z. P. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; García-Dorado, David (Universitat Autònoma de Barcelona. Departament de Medicina) ; Miatello, R. M. (Institute of Medical and Experimental Biology of Cuyo. CONICET) ; Rodríguez-Sinovas, A. (Centro de Investigación Biomédica en Red en Enfermedades Cardiovasculares)