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9 p, 3.7 MB Amyloid-beta peptide and tau protein crosstalk in Alzheimer's disease / Ramos Roda, Alejandro (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular) ; Serra-Mir, Gabriel (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular) ; Montoliu-Gaya, Laia (University of Gothenburg) ; Tiessler, Lidia (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular) ; Villegas Hernández, Sandra (Universitat Autònoma de Barcelona. Departament de Bioquímica i de Biologia Molecular)
Alzheimer's disease is a neurodegenerative disease that accounts for most of the 50-million dementia cases worldwide in 2018. A large amount of evidence supports the amyloid cascade hypothesis, which states that amyloid-beta accumulation triggers tau hyperphosphorylation and aggregation in form of neurofibrillary tangles, and these aggregates lead to inflammation, synaptic impairment, neuronal loss, and thus to cognitive decline and behavioral abnormalities. [...]
2022 - 10.4103/1673-5374.332127
Neural Regeneration Research, Vol. 17, Num. 8 (August 2022) , p. 1666-1674  

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