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Resultats globals: 5 registres trobats en 0.01 segons.
Articles, 5 registres trobats
Articles 5 registres trobats  
1.
12 p, 4.8 MB Down syndrome and leukemia : from basic mechanisms to clinical advances / Baruchel, André (Hôpital Universitaire Robert Debré, Paris, France) ; Bourquin, Jean Pierre (University Children's Hospital, Zurich, Switzerland) ; Crispino, John (St. Jude Children's Research Hospital (Memphis, Estats Units d'Amèrica)) ; Cuartero, Sergi (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Hasle, Henrik (Aarhus University Hospital (Aarhus, Dinamarca)) ; Hitzler, Johann (The Hospital for Sick Children, Toronto, Canada) ; Klusmann, Jan Henning (Goethe University Frankfurt, Germany) ; Izraeli, Shai (Tel Aviv University, Tel Aviv, Israel) ; Lane, Andrew A. (Dana-Farber Cancer Institute (Boston, Estats Units d'Amèrica)) ; Malinge, Sebastien (Cancer Centre, Perth, Western Australia) ; Rabin, Karen R. (University of Oxford, UK) ; Roberts, Irene (University of Oxford, UK) ; Ryeom, Sandra (Columbia University Irving Medical Center, New York, USA) ; Tasian, Sarah K. (University of Pennsylvania Perelman School of Medicine, Philadelphia, USA) ; Wagenblast, Elvin (Icahn School of Medicine at Mount Sinai, New York, USA)
Children with Down syndrome (DS, trisomy 21) are at a significantly higher risk of developing acute leukemia compared to the overall population. Many studies investigating the link between trisomy 21 and leukemia initiation and progression have been conducted over the last two decades. [...]
2023 - 10.3324/haematol.2023.283225
Haematologica, Vol. 108 Núm. 10 (october 2023) , p. 2570-2581  
Registre complet - Creative Commons
2.
33 p, 8.1 MB CEBPA phase separation links transcriptional activity and 3D chromatin hubs / Christou-Kent, Marie (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Cuartero, Sergi (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Garcia-Cabau, Carla (Institute for Research in Biomedicine (IRB Barcelona)) ; Ruehle, Julia (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Naderi, Julian (Department of Genome Regulation. Max Planck Institute for Molecular Genetics) ; Erber, Julia (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Neguembor, Maria Victoria (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Plana-Carmona, Marcos (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Alcoverro Bertran, Marc (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; de Andrés-Aguayo, Luisa (Centre de Regulació Genòmica) ; Klonizakis, Antonios (Centre for Genomic Regulation (CRG). The Barcelona Institute of Science and Technology) ; Julià Vilella, Eric (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Lynch, Cian (Altos Labs. Cambridge Institute of Science) ; Serrano, Manuel (Altos Labs. Cambridge Institute of Science) ; Hnisz, Denes (Department of Genome Regulation. Max Planck Institute for Molecular Genetics) ; Salvatella, Xavier (Institució Catalana de Recerca i Estudis Avançats) ; Graf, Thomas (Universitat Pompeu Fabra) ; Stik, Grégoire (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras)
Cell identity is orchestrated through an interplay between transcription factor (TF) action and genome architecture. The mechanisms used by TFs to shape three-dimensional (3D) genome organization remain incompletely understood. [...]
2023 - 10.1016/j.celrep.2023.112897
Cell reports, Vol. 42 Núm. 8 (29 2023) , p. 112897  
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3.
16 p, 4.9 MB Cohesin couples transcriptional bursting probabilities of inducible enhancers and promoters / Robles-Rebollo, Irene (Imperial College London) ; Cuartero, Sergi (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Canellas-Socias, Adrià (Institut de Recerca Biomèdica) ; Wells, Sarah (Imperial College London) ; Karimi, Mohammad M (King's College London) ; Mereu, Elisabetta (Institut de Ciència i Tecnologia de Barcelona. Institut de Recerca en Biomedicina) ; Chivu, Alexandra G (Cornell University) ; Heyn, Holger (Institut de Ciència i Tecnologia de Barcelona. Institut de Recerca en Biomedicina) ; Whilding, Chad (Imperial College London) ; Dormann, Dirk (Imperial College London) ; Marguerat, Samuel (Imperial College London) ; Rioja, Inmaculada (GlaxoSmithKline Medicines Research Centre (Regne Unit)) ; Prinjha, Rab K (GlaxoSmithKline Medicines Research Centre (Regen Unit)) ; Stumpf, Michael P H (University of Melbourne) ; Fisher, Amanda G (Imperial College London) ; Merkenschlager, Mattihas (Imperial College London) ; Universitat Autònoma de Barcelona
Innate immune responses rely on inducible gene expression programmes which, in contrast to steady-state transcription, are highly dependent on cohesin. Here we address transcriptional parameters underlying this cohesin-dependence by single-molecule RNA-FISH and single-cell RNA-sequencing. [...]
2022 - 10.1038/s41467-022-31192-9
Nature communications, Vol. 13 Núm. 1 (december 2022) , p. 4342  
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4.
18 p, 1.1 MB The mutational landscape of myeloid leukaemia in down syndrome / de Castro, Carini Picardi Morais (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Cadefau, Maria (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Cuartero, Sergi (Institut Germans Trias i Pujol. Hospital Universitari Germans Trias i Pujol) ; Universitat Autònoma de Barcelona
Children with Down syndrome (DS) are particularly prone to haematopoietic disorders. Paediatric myeloid malignancies in DS occur at an unusually high frequency and generally follow a well-defined stepwise clinical evolution. [...]
2021 - 10.3390/cancers13164144
Cancers, Vol. 13 Núm. 16 (august 2021) , p. 4144  
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5.
9 p, 1.3 MB Towards a Better Understanding of Cohesin Mutations in AML / Cuartero, Sergi (Institut Germans Trias i Pujol. Institut de Recerca contra la Leucèmia Josep Carreras) ; Innes, A. J. (Faculty of Medicine. Centre for Haematology. Imperial College London) ; Merkenschlager, M. (Imperial College London. Institute of Clinical Sciences) ; Universitat Autònoma de Barcelona
Classical driver mutations in acute myeloid leukemia (AML) typically affect regulators of cell proliferation, differentiation, and survival. The selective advantage of increased proliferation, improved survival, and reduced differentiation on leukemia progression is immediately obvious. [...]
2019 - 10.3389/fonc.2019.00867
Frontiers in Oncology, Vol. 9 (september 2019) , p. 867  
Registre complet - Creative Commons

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